| 000 | 03210nam a22004217a 4500 | ||
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| 001 | 81238 | ||
| 003 | MX-TxCIM | ||
| 005 | 20170719154814.0 | ||
| 008 | 121211s ||||f| 0 p|p||0|| | | ||
| 020 | _a970-648-116-8 | ||
| 040 | _aMX-TxCIM | ||
| 072 | 0 | _aE16 | |
| 072 | 0 | _aF30 | |
| 090 | _aLook under series title | ||
| 100 | 1 |
_aKumar, R. _uAsian Regional Maize Workshop, 8: New Technologies for the New Millennium _9560 |
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| 110 | 0 | _aCentro Internacional de Mejoramiento de Maiz y Trigo (CIMMYT), Mexico, DF (Mexico) | |
| 111 | 2 |
_aProceedings of the Asian Regional Maize Workshop _cBangkok (Thailand) _d5-8 Aug 2002 |
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| 245 | 0 | 0 | _aInheritance of resistance to banded leaf and sheath blight (Rhizoctonia solani f.sp. sasakii) of maize (Zea mays l.) |
| 260 |
_aMexico, DF (Mexico) _bCIMMYT : _c2004 |
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| 300 | _ap. 356-365 | ||
| 340 | _aPrinted | ||
| 520 | _aOne of the main deterrents to high grain yield in maize is its susceptibility to several diseases. Banded leaf and Sheath blight (BLSB) caused by Rhizoctonia solani .(f. sp. sasakii) has become a serious threat to its cultivation in the north -western plains of India. The pathogen spreads from the basal sheath to the developing ear under favorable environmental conditions. The developing ear is completely damaged and dries up prematurely with caking of husk leaves. The main objective was to work out the inheritance of resistance to this biotic stress. Inheritance of resistance to BLSB was studied based on the analysis of 10 crosses. Eight crosses were between 2 resistant (CM104 and CML1) and 4 susceptible inbred lines (Tarun (x)- 36-1-1-1-2-1-4, D741(x)- 1-1-1-10-4- 4, Pop31 (x)-21-l-2-4-l-l-5-l/13 # and Tarun (x)- 252-1-2-2-1-2-1). In addition, one resistant x resistant and one susceptible x susceptible cross was also studied. Parents, F1's, F2's, and backcrosses, were included in the present study. Final evaluation was undertaken under artificially created epiphytotic conditions. The F2 segregation pattern for BLSB reaction was 15:l in crosses involving CM104 as the resistant parent, and 13:3 in crosses involving CML1 as the resistant parent. The BC1 P1 segregation pattern exhibited a 3:1 ratio. The resistant x resistant and susceptible x susceptible crosses followed all: 0 and 0: all for resistance and susceptibility, respectively. The F2 segregation analysis of eight susceptible x resistant crosses revealed that resistance was governed by two genes. The BLSB reaction in F2 and backcrosses involving CM 104 and susceptible lines suggested that resistance in CM 104 was controlled by duplicate dominant genes while crosses of CML1 showed dominance and recessive interaction. | ||
| 546 | _aEnglish | ||
| 591 | _a0502|AGRIS 0501|AL-Maize Program | ||
| 593 | _aJuan Carlos Mendieta | ||
| 595 | _aCPC | ||
| 650 | 1 | 0 |
_aGenetic resistance _92277 |
| 650 | 1 | 0 |
_aGrain yield _91339 |
| 650 | 1 | 0 | _aInheritance (genetics) |
| 650 | 1 | 0 |
_aMaize _91173 |
| 650 | 1 | 0 |
_aPlant diseases _91206 |
| 650 | 1 | 0 | _aRhizoctonia solani |
| 653 | 0 | _aCIMMYT | |
| 650 | 1 | 0 |
_91314 _aZea mays _gAGROVOC |
| 700 | 1 |
_aSingh, I.S., _ecoaut. |
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| 700 | 1 |
_aSrinivasan, G.|Zaidi, P.H.|Prasanna, B.M.|Gonzalez, F.|Lesnick, K. _eeds. |
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| 942 | _cPRO | ||
| 999 |
_c7473 _d7473 |
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