000			03247nab a22004577a 4500
001			G90305
003			MX-TxCIM
005			20230904205838.0
008			211105s2008 xxu|||p|op||| 00| 0 eng d
022			_a1435-0653 (Online)
024	8		_ahttps://doi.org/10.2135/cropsci2007.04.0185
040			_aMX-TxCIM
041			_aeng
090			_aCIS-5207
100	1		_9473 _aDerera, J.
245	1	0	_aGene action controlling gray leaf spot resistance in southern African maize germplasm
260			_aUSA : _bCSSA : _bWiley, _c2008.
340			_aComputer File|Printed
500			_aPeer review
500			_aPeer-review: Yes - Open Access: Yes|http://science.thomsonreuters.com/cgi-bin/jrnlst/jlresults.cgi?PC=MASTER&ISSN=0011-183X
520			_aGray leaf spot disease (GLS; caused by Cercospora zeae-maydis Tehon and Daniels) is among the major maize (Zea mays L.) production constraints in southern Africa. Maize is predominantly grown by small-scale farmers without fungicides; hence, there is need to develop GLS resistant hybrids. There is limited information about the mode of inheritance for GLS resistance in regionally adapted germplasm. This study was initiated to determine gene action controlling GLS resistance. Seventy-two hybrids were generated by mating 27 inbred lines in a North Carolina design II scheme. Experimental and check hybrids were evaluated in an 8 by 12 α-lattice design with two replications at three locations, during the 2004–2005 season. There was significant variation among the hybrids for GLS resistance and yield. Inbreds L13, L15, L18, L19, and L24, from A, N3, B, K, and SC heterotic groups, respectively, contributed high levels of resistance to hybrids. Both general combining ability (GCA) and specific combining ability (SCA) effects were highly significant (P < 0.01), but the predominance of GCA for GLS (86%) and yield (74%) indicated that additive effects were more important than nonadditive gene action in controlling both traits. Hybrids ranked similarly for GLS across environments, suggesting that few significant crossover genotype by environment interactions, which would cause problems in hybrid selection, were observed. Overall, results indicated that it would be readily possible to develop inbred lines with high GLS resistance from this germplasm.
536			_aGenetic Resources Program|Global Maize Program
546			_aText in English
591			_aCrop Science Society of America (CSSA)
592			_aZA-UKZN 2005 DERERA D r
594			_aINT1617|INT2396
650		7	_2AGROVOC _91206 _aPlant diseases
650		7	_2AGROVOC _91173 _aMaize
650		7	_2AGROVOC _93563 _aGenes
650		7	_2AGROVOC _91077 _aDisease resistance
700	1		_aTongoona, P.B. _8001713456 _gFormerly Excellence in Breeding _9340
700	1		_9832 _aPixley, K.V. _gGenetic Resources Program _8INT1617
700	1		_9837 _aVivek, B. _gGlobal Maize Program _8INT2396
700	1		_aLaing, M.D. _9170
700	1		_avan Rij, N. _924772
740			_a89435
740			_a90305
773	0		_tCrop Science _n635066 _gv. 48, no. 1, p. 93-98 _x1435-0653 _wG444244 _dUSA : CSSA : Wiley, 2008.
856	4		_yAccess only for CIMMYT Staff _uhttps://hdl.handle.net/20.500.12665/970
942			_cJA _2ddc _n0
999			_c26962 _d26962