000 03217nab a22004577a 4500
001 G448292
003 MX-TxCIM
005 20231002202426.0
008 210831s2002 xxu|||p|op||| 00| 0 eng d
022 _a1435-0653 (Online)
024 8 _ahttps://doi.org/10.2135/cropsci2002.1701
040 _aMX-TxCIM
041 _aeng
072 0 _aF30
072 0 _aH20
090 _aCIS-3466
100 1 _aCastro, A.J.
_92689
245 1 0 _aCoincident QTL which determine seedling and adult plant resistance to strip rust in barley
260 _aUSA :
_bCSSA :
_bWiley,
_c2002.
340 _aComputer File
500 _aPeer review
500 _aPeer-review: Yes - Open Access: Yes|http://science.thomsonreuters.com/cgi-bin/jrnlst/jlresults.cgi?PC=MASTER&ISSN=0011-183X
520 _aBarley stripe rust (caused by Puccinia striiformis Westend. f. sp. hordei) is an important disease of barley (Hordeum vulgare L. subsp. vulgare). This disease reached the Americas in 1975. It is now endemic from the Andean region of South America to western North America. We are systematically mapping quantitative resistance genes present in ICARDA/CIMMYT germplasm and introgressing these genes into barley germplasm adapted to western North America. Resistance to stripe rust in the Triticeae can be race- and growth-stage specific. In this study, we mapped genes conferring resistance at the seedling stage, after inoculation with defined isolates (PSH-1, PSH-13, PSH-14), in a doubled haploid population in which adult plant resistance genes had previously been mapped. The disease reaction data for each of three isolates fit a 3:1 (susceptible: resistant) ratio, indicating that two genes are required for resistance. Quantitative trait loci (QTL) effects and significance were estimated by means of QTL mapping procedures and logistic regression analysis, taking into account the binomial distribution of the trait. Two resistance QTL—one on chromosome 5 (5H) and one on chromosome 6 (6H)—were detected and in all cases ‘Shyri’ contributed the resistance alleles. No QTL × race interaction was detected. The two seedling resistance QTL map to the same regions of the genome as two of the four adult plant resistance QTL. These data lay the foundation for more detailed analyses directed at unraveling the genetics of qualitative and quantitative disease resistance mechanisms.
546 _aText in English
591 _a0210|Crop Science Society of America (CSSA)|AL-Wheat Program|Al-ABC Program|R01JOURN
650 7 _aBarley
_91018
_2AGROVOC
650 7 _91842
_aPuccinia striiformis
_2AGROVOC
650 7 _aRusts
_2AGROVOC
_91251
650 7 _aSeedlings
_2AGROVOC
_95415
650 7 _aDisease resistance
_2AGROVOC
_91077
650 7 _aQuantitative Trait Loci
_2AGROVOC
_91853
700 0 _aXianming Chen
_92097
700 1 _aHayes, P.M.
_922669
700 1 _aKnapp, S.J.
_922670
700 1 _aLine, R.F.
_922671
700 1 _aToojinda, T.
_922672
700 1 _aVivar, H.
_95562
773 0 _tCrop Science
_n631533
_gv. 42, no. 5, p. 1701-1708
_dUSA : CSSA : Wiley, 2002.
_wG444244
_x1435-0653
856 4 _yAccess only for CIMMYT Staff
_uhttps://hdl.handle.net/20.500.12665/1323
942 _cJA
_2ddc
_n0
999 _c22477
_d22477