The effects of the deficiency of chromosome V(5B) of Triticum æstivum on the meiosis of synthetic amphiploids
Material type:
ArticleLanguage: English Publication details: 1963. United Kingdom : Springer Nature,ISSN: - 0018-067X
- 1365-2540 (Online)
| Item type | Current library | Collection | Call number | Status | Date due | Barcode | Item holds | |
|---|---|---|---|---|---|---|---|---|
| Article | CIMMYT Knowledge Center: John Woolston Library | Reprints Collection | REP-1156 (Browse shelf(Opens below)) | Available |
Peer-review: Yes - Open Access: Yes|http://science.thomsonreuters.com/cgi-bin/jrnlst/jlresults.cgi?PC=MASTER&ISSN=0018-067X
The chromosomes of the hexaploid wheat of commerce, Triticum&stivum (2n = 6x = 42), form 21 bivalents at meiosis and the occurrence of disomic inheritance demonstrates that every chromosome pairs only with its single homologous partner. However, Riley andChapman (1958) showed that when chromosome V(5B) was absent, from nullisomics or from nullisomic-haploids, there was non-homologous pairing and multivalents were formed. Subsequently it was demonstrated that the non-homologous pairing resulted in recombination between homceologous chromosomes, that is between genetically equivalent chromosomes of the three component genomes of T. rstivum (Riley and Kempanna, 1963). Clearly a genetic activity of chromosome V(5B) is responsible for the suppression of the meiotic affinity of homceologues. No other chromosomes of T. rstivum performs a similar function (Riley, Chapman and Kimber, 1960), and the activity is restricted to the long arm of chromosome V(5B) (Riley, 1960).
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