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Recent advances in the understanding of Fusarium trichothecene mycotoxicoses

By: Smith, T.K.
Material type: materialTypeLabelArticlePublisher: 1992ISSN: 0021-8812.Subject(s): ANIMAL MORPHOLOGY | Central nervous system | Deuteromycotina | Digestive system | Fungi | Miscellaneous animal disorders | Mycotoxins | Nervous system | Toxic substances | ToxinsDDC classification: 93-119086 In: Journal of Animal Science v. 70, no. 12, p. 3989-399393-119086Summary: Recent concepts in the etiology of Fusarium trichothecene mycotoxicoses have been reviewed. The effect of orally administered trichothecenes on tissue metabolism has been traced from the gastrointestinal tract to the liver and subsequently to blood. It is proposed that the hyperaminoacidemia resulting from trichothecene toxicoses contributes to the behavioral changes observed, including loss of appetite and vomiting. Studies with several species and several trichothecenes have shown that elevated brain tryptophan arising from trichothecene-induced aminoacidemia can subsequently alter regional brain serotonin concentrations. This may produceCollection: AGRIS Collection
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Article CIMMYT Knowledge Center: John Woolston Library

Lic. Jose Juan Caballero Flores

 

AGRIS Collection 93-119086 (Browse shelf) 1 Available 93-119086
Total holds: 0

Peer-review: Yes - Open Access: Yes|http://science.thomsonreuters.com/cgi-bin/jrnlst/jlresults.cgi?PC=MASTER&ISSN=0021-8812

references. Paper presented at a symposium entitled "Current Perspectives on Mycotoxins in Animal Feeds," Laramie, WY. Literature review

Recent concepts in the etiology of Fusarium trichothecene mycotoxicoses have been reviewed. The effect of orally administered trichothecenes on tissue metabolism has been traced from the gastrointestinal tract to the liver and subsequently to blood. It is proposed that the hyperaminoacidemia resulting from trichothecene toxicoses contributes to the behavioral changes observed, including loss of appetite and vomiting. Studies with several species and several trichothecenes have shown that elevated brain tryptophan arising from trichothecene-induced aminoacidemia can subsequently alter regional brain serotonin concentrations. This may produce

English

US (DNAL 49 J82)|COMOD

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